Hello guys! I am ecstatic about finding this message board. I’m currently an undergrad human biology student in England, hoping to start a phd in proteomics.
I’m currently in the process of creating a general presentation about a chosen topic (I chose viruses). I have explained the lytic and lysogenic cycles and wanted to mention the reproduction process of retroviruses with the assistance of reverse transcriptase. Taking the HIV virus as an example, upon completion of viral proteins being synthesised inside the host cell, the mature virus can then leave via budding – forming a viral envelope. I know this means that the capsid is enveloped within a portion of the host cell, but does this mean it is now ignored by the immune system as a foreign body because they appear as self-antigens? Or does it just mean it enables faster attachment to non-infected cell proteins to speed up the process of reproduction?
Thanks guys. Looking forward to reading your responses as well as browsing other topics in this field.
Hola,early viral genes made that in the membrane of infected cells would appears protein molecules which facilitate anchoring of virions to new non infected cells spreading infections.when lysis of infected cell is produced , head of virions carries ,in their head ,membrane fragments with the viral recognition proteins, to infect new cells. These structures are known as peplomers and I believe that this is a commun phenomenom in viruses infection. Buena suerte