I may as well make a list out of this!

– Cell death can be due to phagocytosis or by the release of membrane perforating enzymes such as granzyme and perforin – both of these from NK or cytotoxic T-cells – that cause apoptosis.
– Complement directly leads to the destruction of cells by causing a membrane attack complex to be formed and leading to apoptosis by creating a pore in the membrane. Antibodies can be used for tagging, but more often they just bind receptors so they’re useless or cause agglutination of invading particles etc.
– Cytotoxic T-cells cause cell death as above, and helper T-cells collect foreign antigens so that peptide fragments from them can be presented in conjunction with MHC complexes. This allows the maturation and activation of other immune components like B-cells.
– B-cells can differentiate into plasma cells (produce antibodies), memory cells (form germinal centres in the secondary lymphoid follicles) or B1 cells (similar role to plasma cells). Each has a single unique surface receptor.
– CD4 and CD8 are surface glycoproteins, so not really similar to antibodies. They act as co-receptors for the TCR along with MHC, the primary function of which is to hold the two cells close together during recognition.

You’re welcome 🙂

It is a system of biological structures that defend against the infections or disease through discovering and assassinating pathogens and tumor cells…. : 🙄